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Anonymous
Jul 12

briefly explain how bacteria develop resistance to antibiotics and how humans can contribute to the phenomenon.?

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ChatGPT
Jul 12
Bacteria can develop resistance to antibiotics through several mechanisms, including mutations in their genetic material that make them less susceptible to the effects of the drug, acquiring resistance genes from other bacteria through horizontal gene transfer, and forming biofilms that protect them from the antibiotic.

Humans can contribute to the development of antibiotic resistance by overusing antibiotics, not completing the full course of prescribed antibiotics, using antibiotics for non-bacterial infections (such as viral infections), and using antibiotics in agriculture and livestock farming. These practices create selective pressure that allows resistant bacteria to survive and multiply, leading to the spread of antibiotic-resistant strains.
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Google Gemini AI
Jul 12
Bacterial Resistance to Antibiotics

Bacteria have an inherent capacity to evolve and adapt to environmental changes, including antibiotic exposure. This process occurs through:

- Natural selection: Antibiotics kill susceptible bacteria, leaving resistant ones to survive and reproduce.
- Horizontal gene transfer: Resistant bacteria can transfer genetic material encoding resistance genes to other bacteria through mechanisms like plasmids and viruses.

Human Contribution to Antibiotic Resistance

Humans contribute to antibiotic resistance through:

- Overuse and misuse: Unnecessary antibiotic use for viral infections and minor ailments promotes resistance development.
- Incomplete treatment courses: Stopping antibiotic use before completion gives bacteria a chance to recover and develop resistance.
- Animal agriculture: Antibiotics are widely used in livestock to prevent and treat infections, contributing to resistance spread.
- Poor hygiene: Inadequate sanitation and hygiene practices allow resistant bacteria to persist and spread in hospitals and communities.
- Delayed diagnosis: Identifying and treating infections promptly reduces the selective pressure for antibiotic-resistant strains.
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